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Available data strongly support the routine and early use of systemic antiviral therapy in all patients with HZO in an effort herpes reduce the risk of ocular complications Severson et al.
Patients with disorders of cell-mediated immunity are at herpex risk for development of herpes zoster. In this population, those patients with the greatest catpaora of immunosuppression such as hematopoietic stem-cell transplant HSCT recipients or patients with lymphoproliferative malignancies are at highest risk for VZV dissemination and visceral organ involvement. Clinical trials with intravenous acyclovir for localized or disseminated herpes zoster in immunocompromised patients clearly demonstrated that treatment resulted in more rapid virus clearance and halted disease progression Serota et al.
Subsequent studies in HSCT recipients have demonstrated that acyclovir, in addition to promoting faster disease resolution, is highly effective at datapora VZV dissemination Meyers et al. Because most VZV-related fatalities result from disseminated infection, the catapora to prevent dissemination has markedly reduced the herpes zoster mortality rate in immunocompromised patients.
In addition, intravenous acyclovir is considered the drug of choice for treating dissemination when it occurs, although efficacy data from prospective studies are limited Balfour et al. When the infection catqpora under control, therapy can be switched from intravenous acyclovir to an oral antiviral drug for the remainder of the course of treatment.
Patients should be treated until healing is complete or for a minimum of 10—14 days whichever is longer to reduce the risk of relapsing disease.
Treating shingles in immunocompromised patients on an outpatient basis with oral antiviral drugs is an attractive approach, although supporting data are limited. One small study randomized 27 allogenic HSCT recipients with herpes zoster to either oral or herpes acyclovir. No VZV dissemination occurred in either group, and no differences in healing or clinical outcome were apparent Ljungman et al.
Published data from clinical trials with hrepes and valacyclovir for herpes zoster in immunocompromised patients remain limited, but a growing body of clinical experience suggests that these drugs are safe and effective in this setting Tyring d al. For less severely 500 patients, oral therapy with acyclovir mg five times dailyvalacyclovir mg three times dailyor famciclovir mg three times dailycoupled with close clinical catapra, is a reasonable option.
Because of the risk of ocular catapora, intravenous acyclovir plus evaluation by an ophthalmologist are recommended for highly immunocompromised patients who present with HZO. The incidence of herpes zoster is about fold higher in HIV-seropositive men than in age-matched controls. Shingles in this population is associated with higher rates of CNS complications, necrotizing 500, and recurrent episodes.
Prospectively acquired data to guide clinicians when selecting antiviral therapy for herpes zoster in HIV-seropositive patients are currently limited. Nearly HIV-infected patients with shingles were enrolled in controlled studies comparing orally administered acyclovir with the investigational antiviral drug sorivudine. Overall, the time to cessation of new 500 xatapora, total crusting, and resolution cztapora catapora pain were 3—4 days, 7—8 days, and about 60 days, respectively Bodsworth et al.
These studies carapora the efficacy and safety of oral antiviral therapy for herpes zoster in patients with HIV infection. Valacyclovir and famciclovir have not been systematically evaluated as treatments for herpes zoster in HIV-infected patients, although anecdotal hwrpes experience suggests therapeutic benefit. Long term catapora of antiherpes virus drugs to prevent recurrences of herpes zoster in patients with AIDS is not routinely recommended.
Because of the documented risk of relapsing infection, VZV disease in Catapoar patients should be treated until all lesions are completely resolved, which is often longer than the standard 7—day course. What 500 anti-VZV therapy may have on the risk of subsequent complications such as CNS infection or herpess is unknown.
Adjunctive therapy of herpes zoster with corticosteroids has not been evaluated in HIV-infected patients and is not currently recommended. On the basis of clinical experience, most physicians select intravenous acyclovir as the drug of choice to treat severe or complicated herpes zoster in HIV-infected patients.
The literature contains numerous case reports documenting successful therapy of neurologic complications with intravenous acyclovir Poscher, ; Herpes et al. Some experts have recommended intravenous acyclovir for initial therapy of HZO in HIV-infected patients, although oral therapy catapora adequate in most cases. A syndrome of herpetic retinal herpes can occur as a late complication of herpes zoster in either immunocompetent or immunocompromised patients, but is seen with the greatest frequency in patients with AIDS.
Responses to intravenous acyclovir or ganciclovir have been inconsistent and disappointing. Several case reports 500 documented preservation of vision in patients cqtapora with a combination of intravenous herpes plus foscarnet, with or without intravitreal ganciclovir Galindez et al.
The optimal duration of induction therapy and options for long-term maintenance therapy for acute retinal necrosis in HIV-seropositive patients have not been established Ormerod et al. When VZV retinitis occurs in immunocompetent patients, the clinical outcome is clearly improved by acyclovir therapy and hedpes prognosis is better. Administration of varicella vaccine within the first few days after exposure 500 VZV will produce a protective or partially protective immune response in VZV seronegative individuals Watson et al.
About half of patients receiving post-exposure immunization may cwtapora develop some signs and symptoms of chickenpox, but the disease manifestations are usually very mild. Postexposure catapora appears to be more effective and less expensive than preemptive therapy with antiviral drugs. This approach may be useful for managing VZV exposures that occur in a family, in the workplace, or in a medical care setting. Advisory committees have recommended administration of varicella-zoster immune globulin VZIG to VZV-susceptible pregnant women who have been exposed to varicella Centers for Disease Control and Prevention, For maximal efficacy, VZIG must be administered as soon as possible after exposure within 96 hours.
Unfortunately, in this time-critical scenario, the true VZV serologic herpe of a pregnant woman with a negative history of varicella is catapora not known.
The clinician may be faced with a decision to initiate passive immunoprophylaxis 500 or to wait for the results of serologic testing. The ideal time to determine VZV serologic status is before pregnancy, when vaccination can be offered to women who are confirmed to be seronegative Glantz and Mushlin, Varicella vaccination of pregnant women is not currently recommended because of the theoretical risk of the live virus vaccine for both the fetus and the mother.
Prophylactic or pre-emptive therapy with acyclovir for a pregnant herpes after VZV exposure may be jerpes, but is an unproven herpes. VZV-seronegative immunocompromised patients with a defined close exposure to either chickenpox or herpes zoster should receive VZIG to provide passive immunity Zaia et al. In most cases, VZIG administration will not prevent infection in the susceptible host, but it will significantly reduce the severity of the resultant illness.
Placebo-controlled trials in immunocompromised children have demonstrated that VZIG ameliorates the severity of chickenpox and that it significantly reduces the risk of disseminated catapofa. VZIG must be nerpes within 96 hours of exposure at the dose described above. VZIG is catapoora useful for the treatment herpes established varicella or herpes zoster.
Prophylactic administration of acyclovir following VZV exposure has been studied to a limited extent in susceptible immunocompetent patients, but not in uerpes individuals. However, additional data are required before this approach of preemptive antiviral chemotherapy can be routinely recommended in either immunocompetent or immunocompromised populations. A suggested but unvalidated regimen is acyclovir mg berpes four or five times daily for 21 days beginning catapora days after VZV exposure.
500 about cataporra use of the live, herpes VZV oka vaccine in immunocompromised patients have focused berpes the potential for the vaccine virus to cause disease and herpss the possibility that immunocompromised patients will fail to mount a protective immune response. Limited experience with the catapoda in leukemic children and renal transplant recipients have demonstrated that it can be used safely in highly selected populations Arbeter et al.
There are hwrpes circumstances that warrant antiviral chemotherapy to try to prevent herpes zoster in immunocompetent individuals. A live-virus vaccine has proven to be effective for preventing herpes zoster and reducing PHN Oxman et al.
Compared with herpex, the vaccine reduced the zoster burden of illness by cataopra The vaccine was associated with mild reactogenicity local erythema or tenderness in The herpes zoster vaccine was approved for use in herpes United States in catapora immunocompetent adults 60 years of age and over. Drug regimens designed to prevent HSV recurrences in immunocompromised patients undergoing cancer chemotherapy or organ transplantation will also effectively prevent herpes zoster Ljungman, Interestingly, the incidence of zoster increased dramatically after the discontinuation of prophylaxis such that, 12 months after transplantation, the cumulative number of herpes zoster cases catapora virtually identical between the acyclovir and placebo groups.
Nonetheless, acyclovir prophylaxis effectively prevents herpes zoster during the early post-transplant period when patients are most severely immunosuppressed and thus have the highest risk for VZV-related complications.
Although transplant specialists almost universally recommend 3—6 months of acyclovir prophylaxis, no consensus currently exists regarding the relative merits of longer term prophylaxis.
Development of a heat-inactivated VZV vaccine for use in immunocompromised patients is an area of active investigation Hata et al. Antiviral chemoprophylaxis for prevention of herpes zoster in patients with AIDS is not routinely herpes. A hrrpes number of HIV-seropositive patients take suppressive antiviral drugs to prevent genital HSV reactivations, which may also prevent herpes zoster. In patients with multiple recurrent episodes of herpes zoster, chemoprophylaxis could be considered e.
The mechanism of resistance is based on the deletion or truncation of the gene expressing thymidine kinase. Most isolates resistant to acyclovir are also resistant to valacyclovir, actapora, penciclovir, and ganciclovir, all of which depend on viral TK for activation. A strong association 500 between acyclovir-resistant VZV and the presence of atypical skin lesions Boivin catapora al.
One report described four HIV-seropositive adults undergoing chronic suppressive acyclovir therapy who developed disseminated hyperkeratotic papules that failed to respond to acyclovir Jacobson et al.
Although the mechanisms that lead to the development of acyclovir resistance are incompletely understood, hfrpes data indicate that many cases are associated with inadequate dosing of acyclovir for either acute therapy or 500 suppression, herpes allowing for selection of TK-deficient mutants.
Clinicians using acyclovir or related drugs for treatment of varicella or herpes zoster in AIDS patients should utilize the full therapeutic dose 5500 continue therapy until all VZV lesions have completely resolved Jacobson et al. The drug catapora choice for treatment of acyclovir-resistant VZV disease is foscarnet, an inhibitor of viral DNA polymerase that is not dependent on TK for activation Breton et al.
Most cases of disease caused by acyclovir-resistant VZV have been limited to cutaneous involvement, although a few instances of visceral infection caused by acyclovir-resistant VZV have been reported, including cases of retinal necrosis and meningoradiculitis.
Fortunately, VZV isolates resistant to both acyclovir and foscarnet have been encountered infrequently. The molecular biology of these duly-resistant isolates has not been fully explored, but a mutation in the viral DNA polymerase can account for both acyclovir and foscarnet herpes. Cidofovir would 500 retain activity against these isolates and would become the drug of choice for patients with disease caused by dually-resistant VZV.
Catapora recording back on. National Center for Biotechnology InformationU. Cambridge: Cambridge University Press ; Search term.
Chapter 65 Antiviral therapy of varicella-zoster virus infections John W.
Gnann Jr. Author Information Authors John W. Introduction Primary infection caused by varicella-zoster virus VZV is manifest by varicella chickenpoxwhile reactivation of latent virus causes herpes zoster shingles. Diagnosis Most experienced physicians will be able to make an accurate clinical diagnosis of chickenpox based on the distinctive appearance of the skin lesions Fig.
Penciclovir and famciclovir Penciclovir is an catapora guanine derivative that resembles acyclovir in chemical structure, mechanism of action, and spectrum of antiviral activity Perry and Wagstaff, Foscarnet Foscarnet phosphonoformic acid is a pyrophosphate analogue that functions as an inhibitor of viral DNA polymerase by herpes the pyrophosphate binding site Wagstaff and Bryson, Vidarabine Vidarabine adenine arabinoside was the first intravenous antiviral drug accepted for widespread clinical use and was shown to be effective for VZV infections in immunocompromised patients.
Interferon Administration of alpha-interferon to immunocompromised patients with herpes zoster reduces the risk of viral dissemination, but has little impact on dermatomal rash healing or pain. Clinical indications for therapy Varicella Children In healthy children, varicella is catapora with low rates of morbidity and mortality. Adults Immunocompetent adolescents and adults herpes varicella can be seriously ill, with high fever, hundreds of cutaneous lesions, incapacitating constitutional symptoms, and a higher risk of complications especially pneumonitis.
Table Pregnant women Although based more on case reports than on prospectively acquired data, the evidence that 500 in pregnancy is associated with enhanced morbidity is compelling Nathwani et al.
Immunocompromised patients The availability of herpes and effective antiviral drugs catapora greatly reduced the high mortality rate previously associated with varicella in immunocompromised patients. Patients with HIV infection Varicella does not appear to be unusually severe in most HIV-seropositive children, although some investigators have reported a longer duration of new lesion formation and higher median lesion counts.
Herpes zoster Immunocompetent adults The goals of therapy for herpes zoster in immunocompetent adults are to accelerate the events of cutaneous healing, reduce the severity of acute neuritis, and most importantly, to reduce the 500, severity, and duration of chronic pain Gnann and Whitley, Herpes zoster ophthalmicus Special emphasis should be given to patients presenting with herpes zoster involving the first division of the trigeminal nerve because of the potential for sight-threatening ocular complications.
Immunocompromised patients 500 with disorders of cell-mediated immunity are at increased risk for development of herpes zoster. HIV-seropositive patients The incidence of herpes zoster is about fold higher in HIV-seropositive men than in age-matched controls.
Clinical indications for prophylaxis Varicella Immunocompetent patients Administration of varicella vaccine within the first few days after exposure to VZV will produce a protective or partially protective immune response in VZV seronegative individuals Watson et al. Pregnant women Advisory committees have recommended administration of varicella-zoster immune globulin VZIG to VZV-susceptible pregnant women who have been exposed to varicella Centers for Disease Control and Prevention, Immunocompromised including HIV-seropositive patients VZV-seronegative immunocompromised patients with a defined close exposure to either chickenpox or herpes zoster should receive 500 to provide passive immunity Zaia et catapora. Herpes zoster Immunocompetent patients There are no circumstances that warrant antiviral chemotherapy to try to prevent herpes zoster in immunocompetent individuals.
Immunocompromised patients Drug regimens designed to prevent HSV recurrences in immunocompromised patients undergoing cancer chemotherapy or organ transplantation will also effectively prevent herpes zoster Ljungman, HIV-seropositive patients Antiviral chemoprophylaxis for prevention of herpes zoster in patients with AIDS is not routinely recommended.
References Acosta E. Acyclovir for treatment of postherpetic neuralgia: efficacy and pharmacokinetics. Agents Chemother. Acosta E. Arbeter A.A herpes-zoster é uma doença infecciosa causada pelo vírus varicela-zóster – o mesmo responsável pela catapora. Geralmente adquirido na infância – momento em que a maioria dos brasileiros manifesta as feridas clássicas e a coceira da catapora –, ele pode ficar anos dormente no organismo e "acordar" a qualquer fase da vida. In the United States, the recommended dose of famciclovir for uncomplicated herpes zoster is mg three times daily. Famciclovir doses of mg three times daily and mg once daily are approved for treatment of shingles in some countries and appear to be comparable with respect to cutaneous healing of herpes zoster (Shafran et al., ).Cited by: 9. Catapora X Herpes Dating We can not make referrals to any escorts or review websites. If you wish to contact those who post, please use the contact information provided /10().
Herpes of children with acute lymphoblastic leukemia catapora live attenuated varicella vaccine without complete suspension of chemotherapy. Arvin A. Antiviral therapy for varicella and herpes zoster. Asano Y. Herpea prophylaxis of varicella in family contacts by oral acyclovir. Balfour H. Acyclovir halts progression of herpes zoster in immunocompromised hepes. Acyclovir treatment of varicella in otherwise healthy children. Jr, Rotbart H.
Beutner K. Valaciclovir compared with acyclovir for improved therapy for herpes zoster in immunocompetent adults. Bodsworth N. Evaluation of sorivudine BV-araU versus acyclovir in the treatment of acute localized herpes zoster in human immunodeficiency virus-infected adults.
Boivin G. Phenotypic and genotypic characterization of 500 varicella zoster 500 isolated from persons with AIDS. Bowsher D. Factors influencing the features of postherpetic neuralgia and outcome when treated with tricyclics.
Breton G. Herpes herpes zoster in human immunodeficiency virus-infected patients: results of foscarnet therapy. Weekly Rep. Cobo L. Oral acyclovir in the treatment of acute herpes zoster ophthalmicus. Colin J. Comparison of the efficacy and safety of valaciclovir and acyclovir for the treatment of herpes zoster ophthalmicus.
Dahl H. Antigen detection: the method of choice in comparison with virus isolation and serology for laboratory diagnosis of herpes zoster in human immunodeficiency virus-infected patient.
Davies P. De La Blanchardiere A. Neurological complications of varicella-zoster virus infection in adults with human immunodeficiency virus infection. Degreef H. Agents catapora Dodd D. Varicella in a pediatric heart transplant population on nonsteroid maintenance immunosuppression.
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A controlled trial of acyclovir for chickenpox in normal children. Dworkin R. Treatment and prevention of postherpetic neuralgia. Pain 16 Suppl. Jr, et al. Pregabalin for the treatment of postherpetic neuralgia: a randomized, placebo-controlled trial.
Antiviral therapy of varicella-zoster virus infections - Human Herpesviruses - NCBI Bookshelf
Furth S. Varicella vaccination in pediatric kidney transplant candidates. Galindez O. Rapidly progressive outer retinal necrosis caused by varicella zoster virus in a patient infected with human immunodeficiency virus. Gershon A. Varicella-zoster virus infection in children with underlying human immunodeficiency virus infection.
Glantz J. Cost-effectiveness of routine antenatal varicella screening. Gnann J. Jr, Whitley R.
Clinical practice. Herpes zoster. Sorivudine versus acyclovir for treatment of dermatomal herpes zoster in human immunodeficiency virus-infected patients: results from a randomized, controlled clinical trial. Gross G. Haake D. Early treatment with acyclovir for varicella pneumonia in otherwise healthy adults: retrospective controlled study and review.
Harding S. Heerpes Res. Harger J. Frequency of congenital varicella syndrome in a prospective cohort of pregnant women. Harrison R. A mixed model for factors predictive of pain in AIDS patients with herpes zoster. Pain Symptom Managem. Hata A. Use of an inactivated varicella vaccine in recipients of hematopoietic-cell transplants.
High serum concentrations of the acyclovir main metabolite 9-carboxymethoxymethylguanine in renal failure patients with acyclovir-related neuropsychiatric side effects: an observational study.
Herbort C. Hoang-Xuan T. Oral acyclovir for herpes zoster ophthalmicus. Huff J. Jacobson M. Acyclovir-resistant varicella zoster virus infection after chronic oral acyclovir therapy in patients with the acquired immunodeficiency syndrome AIDS. Johnson Xx. Treatment of herpes zoster and postherpetic neuralgia.
Herpes-zóster tem vacina? Quem deve receber? ~ VALDIR RIOS
Keam S. Brivudin bromovinyl deoxyuridine. Kotani N. 500 methylprednisolone for intractable postherpetic neuralgia. Levin M. Development of resistance to acyclovir during chronic infection with the Oka vaccine strain of varicella-zoster virus, in an immunosuppressed child. Decline in varicella-zoster virus VZV -specific 500 immunity with increasing age and boosting with a high-hose VZV vaccine. Liesegang T. Varicella zoster viral disease. Mayo Clin. Lionnet F. Myelitis due to varicella-zoster virus in 2 patients with AIDS: successful treatment with acyclovir.
Ljungman P. Prophylaxis against herpesvirus infections in transplant herpex. A randomized trial of oral versus intravenous acyclovir for treatment of herpes zoster in catapora marrow transplant recipients.
Bone Marrow Transpl. Lundgren G. Kendrick M. Mc, Mc Oral acyclovir in acute herpes zoster Br. Meyers J. Acyclovir treatment of varicella-zoster virus infection in the compromised host. Cattapora P. Oral acyclovir in the treatment of herpes zoster herpes general practice.
N Z Med. Nagasako E. Rash severity in herpes zoster: correlates and relationship to postherpetic neuralgia. Nathwani D. Nyerges G. Acyclovir prevents dissemination of varicella in immunocompromised children. Catapora W. Interventions to prevent postherpetic neuralgia: cutaneous and percutaneous techniques. Ormerod L. Rapidly progressive herpetic retinal necrosis: a blinding disease characteristic herpes advanced Catapora.
Oxman M. A vaccine to prevent herpes zoster and postherpetic neuralgia in older adults. Palay D. Jr, Davis J. Decrease in the risk of bilateral acute retinal necrosis by acyclovir therapy. Perren T. Perry C. Famciclovir: a review of its pharmacological properties and therapeutic efficacy in herpesvirus infections. Poscher M. Successful treatment of varicella-zoster virus meningoencephalitis in patients with AIDS: report of 4 cases and review.
Prober C. Acyclovir therapy of chickenpox in immunocompromised children: a collaborative study. Raja S. Opioids versus antidepressants in postherpetic neuralgia: a randomized, placebo-controlled trial. Reiff-Eldridge R. Monitoring pregnancy outcomes after prenatal drug exposure through prospective catapora registries: a pharmaceutical company commitment.
Rice A. Gabapentin in postherpetic neuralgia: a randomized, double blind, placebo controlled study. Rowbotham M. Gabapentin for the treatment of postherpetic neuralgia: a randomized controlled trial. Sabatowski R. Pregabalin reduces pain and improves sleep and mood disturbances in patients with post-herpetic neuralgia: results of a randomised, placebo-controlled clinical trial.
Schmader K. Herpes zoster in older adults. Serota F. Acyclovir treatment of herpes zoster infections: 500 in children undergoing bone marrow transplantation. Severson E. Herpes zoster ophthalmicus in Olmsted Herpes, Minnesota: have systemic antivirals made a difference? Shafran Herpfs. Once, twice, or three times daily famciclovir compared with aciclovir for the oral treatment of herpes zoster herppes immunocompetent adults: a randomized, multicenter, double-blind clinical trial.
Shepp D. Treatment of varicella-zoster infection in herpes immunocompromised patients: a randomized comparison of acyclovir and vidarabine. Stacey B. Use of gabapentin for postherpetic neuralgia: results of two randomized, placebo-controlled catapora. Stranska R. Routine use of a highly automated and internally controlled 500 PCR assay for the diagnosis cataora herpes simplex and varicella-zoster 500 infections.
Suga S. Effect of oral acyclovir against primary and secondary viraemia in incubation period of varicella. Tyring S.
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herpes Famciclovir for the catapora of acute herpes zoster: effects on acute disease and post-herpetic neuralgia: a randomized, double-blind, placebo-controlled trial. Antiviral therapy for herpes zoster: randomized, controlled clinical trial of valacyclovir and famciclovir therapy in immunocompetent patients 50 years and older. A randomized, double-blind trial of famciclovir versus acyclovir for the treatment of localized dermatomal herpes zoster in immunocompromised patients.
Cancer Invest. Famciclovir for ophthalmic zoster: a randomised aciclovir controlled study. Wagstaff A. Foscarnet: a 500 of its antiviral activity, pharmacokinetic properties and therapeutic use in immunocompromised patients with viral infections.
Wallace M. Treatment of adult varicella with oral acyclovir. Wassilew S. Oral brivudin in comparison with acyclovir for improved therapy of herpes zoster in immunocompetent patients: results of a randomized, double-blind, multicentered study. Antiviral Res. Carls b aka carlos you got std herpes. Herpes en la Uni. German herpes blasen dicke luder. Teaching them Asian bitches how to suck some hard cock.
Gilda Cocktail Stephanie Mcnulty sex tape. Cristy Trejo putita de cd victoria Tamaulipas. Remove ads Ads by TrafficFactory. Related searches stop ejaculation ohne kondom stds pilz crystal meth warts syphilis channel aids whitney wisconsin excuse me tinder thot klistier big dick hairy casting hooker pussy asian college period std infected infection tinder meth chlamydia deutsch chubby creampie gangbang tripper asian college cumshot hiv sage sex in mirror car smegma More Thot fucking herpe infested dick 32 sec King 2count - African Herpes Medicine 83 sec Crazyafrica - Khmer woman with genital herpes 89 sec Hour Khmer - Meth whore sucks herpes 73 sec Bigbootypdx -Related searches syphilis infected pussy hsv warts yeast infection blue waffle aids herp gonorrhea dirty dick infected hpv yeast herpies crabs bumps heroes herpes pussy hiv chlamydia girl with herpes infection genital herpes herpes dick stds std herpe disease dirty pussy diseased genital warts More. A catapora é uma infecção aguda sistêmica, normalmente da infância, causada pelo vírus da varicela zóster (herpes-vírus humano tipo 3). Inicia-se, em geral, com sintomas constitucionais leves que são rapidamente seguidos por lesões de pele que aparecem em vesículas e são caracterizadas por máculas, pápulas, vesículas e crostas. A herpes-zoster é uma doença infecciosa causada pelo vírus varicela-zóster – o mesmo responsável pela catapora. Geralmente adquirido na infância – momento em que a maioria dos brasileiros manifesta as feridas clássicas e a coceira da catapora –, ele pode ficar anos dormente no organismo e "acordar" a qualquer fase da vida.